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Loss-of-function mutations in CLDN1 result in neonatal ichthyosis-sclerosing cholangitis syndrome
with C-terminal Human IgG Fc ISSMDMERPGDGKCQPIEIPMCKDIGYNMTRMPNLMGHENQREAAIQLHEFAPLVEYGCHGHLRFFLCSLYAPMCTEQVSTPIPACRVMCEQARLKCSPIMEQFNFKWPDSLDCRKLPNKNDPNYLCMEAPNNGSDEPTRGIEGRMDPKSSDKTHTCPPCPAPELLGGPSVFLFPPKPKDTLMISRTPEVTCVVVDVSHEDPEVKFNWYVDGVEVHNAKTKPREEQYNSTYRVVSVLTVLHQDWLNGKEYKCKVSNKALPAPIEKTISKAKGQPREPQVYTLPPSRDELTKNQVSLTCLVKGFYPSDIAVEWESNGQPENNYKTTPPVLDSDGSFFLYSKLTVDKSRWQQGNVFSCSVMHEALHNHYTQKSLSLSPGK
acquisition of the Th2 phenotype by CD4+T cells
BLAME is a cell surface receptor that is expressed upon activation of macrophages (MΦs) by IFN-γ or bacteria
MMP-7 is often overexpressed and contributes to tumor progression by promoting cell proliferation
TEL1 Recombinant Rabbit mAb (S-3067-60) Size:100μl Loss-of-function mutations in CLDN1 resultProduct Specification Host Rabbit Antigen TEL1 Synonyms Transcription factor ETV6; ETS translocation variant 6; ETS related protein Tel1 (Tel); TEL; ETV6 Immunogen Synthetic Peptide Location Nucleus Accession P41212 Clone Number S 3067 60 Antibody Type Recombinant mAb Isotype IgG Application WB, IHC P, ICC, IF Reactivity Hu, Ms, Rt, Mk Positive Sample Raji, THP 1, U 2 OS, A431, HeLa, NIH 3T3, PC 12, COS 7 Purification Protein A Concentration 0. 5 mg